Fendikind-plus Suspension
Out of Stock
Fendikind Plus Fenbendazole and Ivermectin Oral Suspension, manufactured under the Vetjoy Mankind division of Mankind Pharma Ltd., is a cutting-edge, broad-spectrum antiparasitic oral suspension formulated to address one of the most clinically significant and economically impactful health challenges in both companion animal and livestock management: concurrent infestation by endoparasites and ectoparasites. Single-agent deworming products frequently fail to provide complete parasite coverage because gastrointestinal worms, lungworms, tapeworms, lice, mites, and grubs each require different mechanisms of antiparasitic action to be effectively eliminated. Fendikind Plus resolves this limitation through a synergistic dual-active combination of Fenbendazole, a benzimidazole anthelmintic targeting gastrointestinal and respiratory nematodes as well as cestodes, and Ivermectin, a macrocyclic lactone that targets both internal and external parasites at the neuromuscular level. Together, these two actives kill all stages of the worm life cycle, including larvae and eggs, while extending protection to the skin and coat by eliminating ectoparasites such as mites and lice. Critically, Fendikind Plus is formulated to be safe for use in pregnant animals, with no reported adverse effects on the foetus, making it a trusted choice of veterinary professionals across livestock and companion animal practice. Animeal is proud to offer Fendikind Plus Oral Suspension as part of our commitment to bringing scientifically validated, veterinarian-approved antiparasitic solutions to every animal under your care.
Ingredients:
Fenbendazole 25mg per ml: Fenbendazole is a broad-spectrum benzimidazole anthelmintic that exerts its antiparasitic action by selectively binding to beta-tubulin, the structural protein subunit responsible for forming microtubules within parasitic cells. Microtubules are essential components of the parasitic cytoskeleton and are required for cell division, intracellular transport, and the structural integrity of the worm's digestive epithelium. By binding to beta-tubulin with high selectivity at a site not present in mammalian cells, Fenbendazole inhibits the polymerisation of tubulin into functional microtubules, disrupting the formation of the mitotic spindle and blocking cellular division in the parasite. This leads to progressive depletion of the endogenous energy reserves of the worm as glucose uptake and glycogen storage are impaired, ultimately causing immobilisation and death of adult parasites, larvae, and arrested larval stages. Fenbendazole is also notably ovicidal, meaning it kills nematode eggs before they can hatch, interrupting the parasite life cycle at its earliest stage and reducing environmental contamination. Its activity spans gastrointestinal nematodes including roundworms (Toxocara spp., Toxascaris leonina), hookworms (Ancylostoma spp., Uncinaria stenocephala), whipworms (Trichuris vulpis), lungworms (Oslerus osleri, Angiostrongylus vasorum), and certain tapeworm species (Taenia spp.) as well as gastrointestinal protozoa such as Giardia spp. at higher doses.
Ivermectin 1mg per ml: Ivermectin is a macrocyclic lactone derived from the fermentation of Streptomyces avermitilis. It acts on the nervous system of invertebrate parasites by selectively binding to and potentiating glutamate-gated chloride ion channels (GluCl channels) in the nerve and muscle cells of nematodes and arthropods. This binding causes an irreversible increase in permeability of the cell membrane to chloride ions, resulting in hyperpolarisation of the nerve or muscle cell. The consequence is prolonged, uncontrolled inhibition of the parasite's neuromuscular transmission, leading to paralysis and rapid death of the organism. Critically, GluCl channels are absent in mammals, and the mammalian blood-brain barrier prevents Ivermectin from accessing GABA-gated chloride channels in the central nervous system at therapeutic doses, making it highly safe in most species. Ivermectin provides comprehensive activity against a wide spectrum of nematodes including gastrointestinal roundworms, lungworms, and microfilariae, as well as ectoparasites including sarcoptic mange mites (Sarcoptes scabiei), psoroptic mange mites (Psoroptes ovis), ear mites (Otodectes cynotis), lice (Damalinia spp., Linognathus spp.), and botfly larvae (Hypoderma spp., Gasterophilus spp.).
